Download PDF by Vittorio E. Andreucci (auth.), Vittorio E. Andreucci (eds.): Acute Renal Failure: Pathophysiology, Prevention, and

By Vittorio E. Andreucci (auth.), Vittorio E. Andreucci (eds.)

ISBN-10: 146129794X

ISBN-13: 9781461297949

ISBN-10: 1461328411

ISBN-13: 9781461328414

`No doubt than it good merits its position within the library of the younger and not more younger nephrologists either in most cases within the medical perform or in research.'
Nephron (1986)
`The ebook is an up to date and invaluable compendium of present wisdom within the medical administration of acute renal failure.'
Journal of the Royal Society of medication, seventy eight (1985)

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Extra info for Acute Renal Failure: Pathophysiology, Prevention, and Treatment

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The magnitude of TGF response to different perfusion rates of distal tubules with Ringer's solution is directly proportional, in normal rats, to juxtaglomerular renin activity {213}. c. The TGF response to marked flow rate changes is significantly reduced in normal rats by i. v. infusion of either All antagonist, Sar\ Ala8 angiotensin II (Saralasin, which interferes with All-receptor interaction) {218}, or converting-enzyme inhibitor SQ 20,881, (which interferes with All formation) {205, 219}. d.

But it remains to be established why indomethacin, which exacerbates glycerol-induced ARF {177} presumably through a reduction in PGE 2 release, does not reduce also TxA2 production since the drug is, indeed, an inhibitor of cyclooxygenase. Thus, either TxA2 is not the only vasoconstricting factor involved in glycerol-induced ARF (more probable hypothesis) or indomethacin does not inhibit PGE2> PGI 2, and TxA2 to the same extent {186}. Further studies using selective inhibition of different pathways of endoperoxide metabolism are therefore necessary {183}.

1·1 rag1ny -1 eu8 -All and sarcosme -1 eu8 All (analogs of All that block the pressor and myotropic activity of All) appeared effective competitive inhibitors of 12sI_AIl binding to the isolated glomeruli [250}. AU receptors in glomeruli seem to differ from those in the renal vasculature [251]. Recent studies have demonstrated a dissociation between intrarenal renin and intrarenal All [7]. Thus, oral sodium loading in rats for three weeks has been shown to suppress PRA and intrarenal renin, as already well known, but to leave unchanged intrarenal All in comparison with rats on normal sodium intake; in sodium deprived rats, instead, a significant inverse linear relationship was observed between intrarenal All and urinary sodium concentration [214}.

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Acute Renal Failure: Pathophysiology, Prevention, and Treatment by Vittorio E. Andreucci (auth.), Vittorio E. Andreucci (eds.)

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